Breast Cancer Is The Number One Reason Affordable Care Act (ACA) Must Stay In Place Congress

Barron’s Medical Journal Reporting From The Texas State Fair In Dallas Texas USA At The Cotton Bowl The Sight For This Year’s Prairie View A&M University Vs. Grambling State University NCAA Football Game.

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Breast Cancer Is The Number One Reason Affordable Care Act (ACA) Must Stay In Place Congress. 

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Dallas Texas ( AP ) Barron’s Medical Journal is in Dallas asking the question is the team and fans of the Prairie View A&M University Vs Grambling State University Game How many people attending the game will have a relatives that will get breast cancer. Research says it is eighteen percent.Affordable Care Act (ACA) by design is to reduce the rate of eighteen percent in the African American Communities to fewer than five percent of patients that will die one year after diagnoses.
Affordable Care Act (ACA) or Obamacare, is a United States federal statute signed into law by President Barack Obama on March 23, 2010. Together with the Health Care and Education Reconciliation Act
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Barron’s Medical Journal Ask Rose Conrad Ph.D. C.E.O Of Sam Houston Biotech to talk about how breast cancer works to destroy a patients life. Conrad says that no one dies of breast cancer only when breast cancer spreads to your lungs. If you get a breast cancer genomics test by the age of forty you have a ninety four percent chance to live to sixty. Conrad gose on to say Breast Cancer and HIF-1 Is Why ObamaCare must stay in place. To stop breast cancer from spreading to the patients lungs. Breast cancer spreading to the lungs is the number one reason breast cancer patients expire. Primary Care Physicians and Obama Care is key to prevent women expiring from breast cancer and extend their life.
Sam Houston Biotechnology Scientist Using Genomics has made progress in Houston working with HIF-1. Hypoxia-inducible factor 1 (HIF-1) is a transcriptional activator forbreast cancer. Advances in biochemical purifica- tion, molecular characterization and cellular - molecular biol- ogy ofHIF-1. Sam Houston found involvement in human breast cancer progression, based on the analysis of human breast cancer biopsies and experimental animal mice models. HIF-1 as a therapeutic target can extend the life of many stage four breast Cancer patients.
Sam Houston discovered The tumor suppressors VHL (von Hippel-Lindau protein) and p53 target HIF-1α for ubiquitination to inactivation breast cancer tumors cells increases the of HIF-1. This process Increased phosphatidylinositol 3-kinase (PI3K) and AKT. In breast cancer, increased activity of the HER2 (also known as neu) receptor tyrosine kinase is associated with increased tumor grade, chemotherapy resistance, and decreased patient survival. HER2 has also been implicated as an inducer of VEGF expression. Sam Houston demonstrate that HER2 signaling induced by overexpression in mouse 3T3 cells or human MCF-7 breast cancer cells results in increased HIF-1α protein and VEGF mRNA expression that is dependent upon activity of PI3K, AKT (also known as protein kinase B), and the downstream kinase FRAP (FKBP-rapamycin-associated protein)
Sam Houston concern with patients with diabetes and breast cancer may not receive full Benefit of HIF-1. All women having access to a physician can have access to HIF-1. Genomics provide a faster cheaper more effective way to detecting Breast Cancer by using Semiconductor Sequencing. A example of this technique is Sam Houston Semiconductor Sequencing. "Quantum Theory" In Action for Breast Cancer Patients.
A polymerase is an enzyme whose central function is associated with polymers of nucleic acids such as RNA and DNA. The primary function of a polymerase is the polymerization of new DNA or RNA against an existing DNA or RNA template in the processes of replication and transcription. In association with a Sam Houston also uses a Visualize Real-Time Breast Cancer Data using Signal Stochastic Resonance Units Neurons Detection and Analysis for Breast Cancer model after McCulloch-Pitts. Gennxeix. computer-assisted diagnosing of breast cancer from mammograms. Sam Houston works is a genetic network simulation trained with tumor incidence data from knockout experiments.
Sam Houston uses Semiconductor Sequencing Chips that create a direct connection between Biochemical and digital information, bringing these two languages together. Gennxeix's chips are designed like any other semiconductor chips.
Pairing proprietary semiconductor technology with sequencing chemistry a nucleotide is incorporated into a strand of DNA by a polymerase, a hydrogen ion is released. Sam Houston used a high-density array of micro-machined wells for bioctechnology process in a massive way. Each well holds a different DNA template. Beneath the wells is an ion-sensitive layer and beneath that a proprietary Ion sensor.
Genomics can be the GPS to Extend life in Breast Cancer Patients.